June 8, 2013
Researchers at the Massachusetts Institute of technology (MIT) have identified the cause of the ease by which recent strains of bird flu are transmitted from person to person – genes.
MIT scientists discovered that 3 HA regions, the proteins that allow the virus to infect humans, have been mutated to enable a more efficient bonding to human receptors.
The mutations in the genes assist the virus in living in the human cells which can be a correlation between an outbreak and a pandemic.
The ability to mutate increases with each jump from person to person.
According to the study: “Every pandemic emergence seems to be a law unto itself. We cannot know whether or under what circumstances the highly unusual H7N9 virus might be able to become pandemic . . . H7N9’s journey has just begun.”
The elderly are at a greater risk from the H7N9. Male patients of an average age of 61 years are being admitted into hospitals for treatment of symptoms.
Doctors at the New England Journal of Medicine (NEJM) are asking whether or not “every avian influenza virus capable of infecting humans can acquire serial pandemic-generating mutations without being limited by structural or functional evolutionary constraints — or whether pandemic viruses are rare entities whose complex gene constellations cannot easily be configured except by rare and still-obscure mechanisms?”
The National Health and Family Planning Commission (NHFPC) in China reports that the H7N9 virus has claimed 35 people. In response, citizens are keeping away from poultry markets and fast food stores like KFC.
Dr. David Swayne, director at the US Department Agriculture (USDA) Southeast Poultry Research Laboratory (SPRL) has discovered that the H7N9 virus infects the bird’s gut, not respiratory system.
Swayne said: “I think that kind of points the finger to say, hey, you know, what’s really going to be important in the field is not thinking about ducks and geese as being the primary source of the virus growing and spreading, its chickens and quail. The data we produced experimentally … would suggest that chickens, possibly quail could be amplifiers of the virus and are causing a lot of environmental contamination. Pigeons appear not to be really … perpetrators in spreading this virus around.”
Dr. Jenny Low, senior consultant at the Department of Infectious Disease, Singapore General Hospital commented : “The avian influenza A (H7N9) is potentially deadly. Producing a vaccine to combat this new bird flu strain may however take several months. The good news is that bird flu does not spread easily from person to person.”
H7N9 can cause symptoms such as:
• Shortness of breath
• Acute respiratory distress syndrome (ARDS)
• Septic shock
• Multiple organ failure
If the H7N9 virus turns out like the H5N1 strain, it will become quite clear that this is an engineered bioweapon.
Speaking about the H5N1 virus, Dr. Thomas Inglesby, a bioterrorism expert and director of the Center for Biosecurity of the University of Pittsburgh Medical Center, said : “It’s just a bad idea for scientists to turn a lethal virus into a lethal and highly contagious virus. And it’s a second bad idea for them to publish how they did it so others can copy it.”
In fact, People’s Liberation Army Senior Colonel Dai Xu publically stated that he blamed the US government for releasing the H7N9 in an act of biological warfare.
Xu wrote in a blog post: “At that time, America was fighting in Iraq and feared that China would take advantage of the opportunity to take other actions. This is why they used bio-psychological weapons against China. All of China fell into turmoil, and that was exactly what the United States wanted. Now, the United States is using the same old trick. China should have learned its lesson and should calmly deal with the problem.”
When attacking a specific race of people, their genetic make-up is first and foremost.
Ironically, British and Chinese researchers have discovered a genetic variant common to Chinese people that make them more susceptible to contracting the swine flu. It was determined that an estimated 25% of Chinese people have this gene variant which is also common to people of Japanese and Korean decent.
Tao Dong, lead author of the study and professor at Oxford University, explained: “Understanding why some people may be worse affected than others is crucial in improving our ability to manage flu epidemics and to prevent people dying from the virus.”
The researchers studied 83 patients who were diagnosed with swine flu during 2009-2010. Those patients who were part of the study had died from complications from pneumonia, respiratory or kidney failure and it was shown that 69% had this genetic variation.
It was found that 1% of Caucasians have this genetic variant.
The findings of these researchers suggest that those with a genetic predisposition to severe flu should seek out treatment immediately to control the effects and mitigate future possible reactions because they would be susceptible to not only the flu, but also SARS.
The original SARS was clearly a genetically created bioweapon . It had spliced elements of human, cow and mouse viruses which is not possible in nature. Coronaviruses do not jump species – like infecting animals and spreading to humans.
Another tell-tale sign is that the original SARS did not match any known virus. At first officials claimed it was an A virus, then a B virus.
US Army Command and General Staff College at Fort Leavenworth, Kansas published information in a journal intended for military personnel called Ethnic Specific Weapons, wherein the history, desirability and possibilities of biological weapons tailored for specific races of human could be genetically engineered to defend a nation against attack.
Dr. William Schaffner, chairman of preventative medicine at Vanderbilt University, explained: “It took the outbreak in Hong Kong and subsequent spread to bring that to our attention. The surveillance for viruses that cause disease, particularly respiratory viruses, has improved enormously over the last 10 years worldwide. What happened here demonstrates that.”